Explore the latest developments concerning Popular drug metformin.
When consent finally learns to remember
Consent has always struck me as one of privacy’s strangest inventions. We have spent years building systems designed to ask human beings questions they do not particularly want to answer, usually at the precise moment they are trying to do something else. A customer wants to read an article, check an investment, transfer money or buy a shirt, yet a rectangle appears asking them to participate in a miniature referendum on advertising technology.
Behind that rectangle sits an extraordinary machine. Consent management platforms, SDKs, advertising identifiers, pixels, preference centres, vendor lists and databases wait for the customer’s finger to make a decision. Somewhere, a privacy professional has spent weeks debating whether “Accept All” is too prominent, whether rejection requires another click, and whether an analytics technology is genuinely necessary or merely wearing the clothes of necessity.
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Popular drug metformin transforms your gut into a sugar sink
Metformin is one of the most prescribed medicines in the U.S., primarily to treat or prevent type 2 diabetes. But how it works has been something of a mystery. Now researchers have discovered that the substance essentially transforms a person's gut cells into a sugar sink, resulting in lowered levels of glucose.
Evidence of this mechanism stretches as far back as 1989, when French metabolic researcher Jean Girard and his colleagues published a paper describing how metformin increased the amount of glucose rats absorbed in their intestines. But when research picked up after the drug was finally approved by the U.S. Food and Drug Administration, in 1995, scientists mostly moved toward the theory that metformin reduced the liver's production of glucose. "It was as if the preexisting science had never existed," says University of Cambridge metabolism researcher Stephen O'Rahilly, who was not involved in the new study.
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